Endocardial Fibroelastosis [EFE] by Lashel Jones RDMS-RDCS (FE)

our weekly ultrapost coming this week from USA 🇺🇸 by my LinkedIn friend Lashel Jones RDMS-RDCS

she was discussing my case of fetal critical aortic stenosis posted this week , link to the video 📹 below

recently went over a case study about EndocardialFibroelastosis (EFE), a condition where the inner lining of theheart thickens and becomes white (hyperechoic) because ofextra collagen and elastic fibers. Here are some main points:Types of EFE:Primary Endocardial FibroelastosisDilated Type: Isolated to the endocardium of ahypertrophied left ventricleContracted Type: Although rare, involves a small leftventricular cavity.Secondary Endocardial Fibroelastosis:⁃ Occurs with certain congenital heart malformations such asaortic stenosis, coarctation of the aorta, anomalous origin ofthe left coronary artery from the pulmonary trunk, andhypoplastic left heart.Clinical Considerations:Outflow Obstruction: A crucial factor in secondary EFE,where outflow obstruction leads to stagnant blood, causingfibrin deposition on the endocardium, often resulting inBRIGHT WALLS on imaging.⁃ Systemic Implications: Endocardial fibrosis provides asubstrate for mural thrombosis, increasing the risk ofsystemic emboli. In some cases, the endocardium may evencalcify.

Implications and Next Steps:Understanding the subtype and etiology of EFE is vital fortargeting treatment strategies. For instance, recognizingoutflow obstruction as a causative factor in secondary EFEcan shape both preventive and interventional approaches,potentially improving patient outcomes